PlantImRecepV1, Main, Exploration, bibRecord, 000164

ER Stress Leaves an Inflammatory TRAIL.

Identifieur interne : 000164 ( Main/Exploration ); précédent : 000163; suivant : 000165

ER Stress Leaves an Inflammatory TRAIL.

Auteurs : Alba Roca Portoles [Royaume-Uni] ; Stephen W G. Tait [Royaume-Uni]

Source :

Developmental cell [ 1878-1551 ] ; 2020.

RBID : pubmed:32208160

Descripteurs français

KwdFr :
- Apoptose (MeSH), Feuilles de plante (MeSH), Humains (MeSH), Inflammation (MeSH), Ligand TRAIL (MeSH), Récepteurs de TRAIL (MeSH), Stress du réticulum endoplasmique (MeSH).
MESH :
- Apoptose, Feuilles de plante, Humains, Inflammation, Ligand TRAIL, Récepteurs de TRAIL, Stress du réticulum endoplasmique.

English descriptors

KwdEn :
- Apoptosis (MeSH), Endoplasmic Reticulum Stress (MeSH), Humans (MeSH), Inflammation (MeSH), Plant Leaves (MeSH), Receptors, TNF-Related Apoptosis-Inducing Ligand (MeSH), TNF-Related Apoptosis-Inducing Ligand (MeSH).
MESH :
- chemical : Receptors, TNF-Related Apoptosis-Inducing Ligand, TNF-Related Apoptosis-Inducing Ligand.
- Apoptosis, Endoplasmic Reticulum Stress, Humans, Inflammation, Plant Leaves.

Abstract

Cell stress often triggers inflammation as a protective response to tissue damage or infection. In this issue of Developmental Cell, Sullivan et al. (2020) reveal an unexpected role for TRAIL receptors in ER-stress-induced inflammation.

DOI: 10.1016/j.devcel.2020.03.004
PubMed: 32208160

Affiliations:

Links toward previous steps (curation, corpus...)

to stream Main, to step Corpus: 000183
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Le document en format XML

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<term>Inflammation (MeSH)</term>
<term>Plant Leaves (MeSH)</term>
<term>Receptors, TNF-Related Apoptosis-Inducing Ligand (MeSH)</term>
<term>TNF-Related Apoptosis-Inducing Ligand (MeSH)</term>
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<keywords scheme="KwdFr" xml:lang="fr"><term>Apoptose (MeSH)</term>
<term>Feuilles de plante (MeSH)</term>
<term>Humains (MeSH)</term>
<term>Inflammation (MeSH)</term>
<term>Ligand TRAIL (MeSH)</term>
<term>Récepteurs de TRAIL (MeSH)</term>
<term>Stress du réticulum endoplasmique (MeSH)</term>
</keywords>
<keywords scheme="MESH" type="chemical" xml:lang="en"><term>Receptors, TNF-Related Apoptosis-Inducing Ligand</term>
<term>TNF-Related Apoptosis-Inducing Ligand</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Apoptosis</term>
<term>Endoplasmic Reticulum Stress</term>
<term>Humans</term>
<term>Inflammation</term>
<term>Plant Leaves</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Apoptose</term>
<term>Feuilles de plante</term>
<term>Humains</term>
<term>Inflammation</term>
<term>Ligand TRAIL</term>
<term>Récepteurs de TRAIL</term>
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<front><div type="abstract" xml:lang="en">Cell stress often triggers inflammation as a protective response to tissue damage or infection. In this issue of Developmental Cell, Sullivan et al. (2020) reveal an unexpected role for TRAIL receptors in ER-stress-induced inflammation.</div>
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<Title>Developmental cell</Title>
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<ArticleTitle>ER Stress Leaves an Inflammatory TRAIL.</ArticleTitle>
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	Serveur d'exploration sur les récepteurs immunitaires végétaux
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Serveur d'exploration sur les récepteurs immunitaires végétaux

ER Stress Leaves an Inflammatory TRAIL.

ER Stress Leaves an Inflammatory TRAIL.

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki